1. Schulte PA, Wagner GR, Ostry A, Blanciforti LA, Cutlip RG, Krajnak KM, et al. Work, obesity, and occupational safety and health. AJPH. 2007; 97(3): 428-36.

2. Mirmiran P, Mirbolooki M, Azizi F. Familial clustering of obesity and the role of nutrition: Tehran Lipid and Glucose Study. IJO. 2002; 26(12): 1617-22.

3. Drong A, Lindgren C, McCarthy M. The genetic and epigenetic basis of type 2 diabetes and obesity. CPT, 2012; 92: 707–15.

4. Kaur J. A comprehensive review on metabolic syndrome. Cardiol Res Pract 2019; 4301528.

5. Popko K, Gorska E, Stelmaszczyk-Emmel A,Plywaczewski R, Stoklosa A, Gorecka D, et al.Proinflammatory cytokines Il-6 and TNF-! and thedevelopment of inflammation in obese subjects. EurJ Med Res 2010; 15: 120-2.

6. Jung UJ, Choi M-S. Obesity and its metabolic complications: the role of adipokines and the relationship between obesity, inflammation, insulin resistance, dyslipidemia and nonalcoholic fatty liver disease. Int J Mol Sci 2014; 15(4): 6184-223.

7. Piya MK, McTernan PG, Kumar S. Adipokine inflammation and insulin resistance: the role of glucose, lipids and endotoxin. J Endocrinol 2013; 216(1): T1-T15.

8. Kishore U, Gaboriaud C, Waters P, Shrive AK, Greenhough TJ, Reid KB, et al. C1q and tumor necrosis factor superfamily: modularity and versatility. Trends Immunol, 2004; 25: 551–61.

9. [9]Schäffler A, Buechler C. CTRP family: linking immunity to metabolism. Trends Endocrinol Metab 2012; 23: 194–204.

10. Maeda T, Abe M, Kurisu K, Jikko A, Furukawa S. Molecular cloning and characterization of a novel gene, CORS26, encoding a putative secretory protein and its possible involvement in skeletal development. JBC 2001; 276(5): 3628-34.

11. Wurm S, Neumeier M, Weigert J, Schäffler A, Buechler C. Plasma levels of leptin, omentin, collagenous repeat-containing sequence of 26-kDa protein (CORS-26) and adiponectin before and after oral glucose uptake in slim adults. Cardiovascular Diabetol 2007; 6(1):7.

12. Jung UJ, Choi MS. Obesity and its metabolic complications: the role of adipokines and the relationship between obesity, inflammation, insulin resistance, dyslipidemia and nonalcoholic fatty liver disease. Int J Mol Sci 2014; 15: 6184–223.

13. Li Y, Wright GL, Peterson JM. C1q/TNF-related protein 3 (CTRP3) function and regulation. Compr Physiol 2017; 7(3): 863-78.

14. Fadaei R, Moradi N, Baratchian M, Aghajani H, Malek M, Fazaeli AA, et al. Association of C1q/TNF-Related Protein-3 (CTRP3) and CTRP13 Serum Levels with Coronary Artery Disease in Subjects with and without Type 2 Diabetes Mellitus. PloS One 2016; 11: e0168773.

15. Ahmed SF, Shabayek MI, Abdel Ghany ME, El-Hefnawy MH, El-Mesallamy HO. Role of CTRP3, CTRP9 and MCP-1 for the evaluation of T2DM associated coronary artery disease in Egyptian postmenopausal females. PLoS ONE 2018; 13(12): e0208038.

16. Peterson JM, Wei Z, Wong GW. C1q/TNF-relatedprotein-3 (CTRP3), a novel adipokine that regulateshepatic glucose output. J Biol Chem 2010; 285:39691-701.

17. Kopp A, Bala M, Buechler C, Falk W, Gross P, Neumeier M, et al. C1q/TNF-related protein-3 represents a novel and endogenous lipopolysaccharide antagonist of the adipose tissue. Endocrinology 2010; 151: 5267–78.

18. Deng W, Li C, Zhang Y, Zhao J, Yang M, Tian M, Li L, Zheng Y,Chen B, Yang G. Serum C1q/TNF-related protein-3 (CTRP3) levels aredecreased in obesity and hypertension and are negatively correlated withparameters of insulin resistance. Diabetol Metab Syndr 2015; 7: 33.

19. Azali Alamdari K, khalafi M, Ghorbanian B. Effect of Aerobic Training on Serum Adiponectin and Ctrp-3 in Males with Metabolic Syndrome. IJEM 2017; 18 (5): 368-77. [in persian].

20. Choi HY, Park JW, Lee N, Hwang SY, Cho GJ, Hong HC, et al. Effects of a combined aerobic and resistance exercise program on C1q/TNF-related protein-3 (CTRP-3) and CTRP-5 levels. Diabetes Care 2013; 36(10): 3321–7.

21. Hasegawa N, Fujie S, Horii N, Uchida M, Kurihara T, SanadaK, et al. Aerobic exercise training-inducedchanges in serum C1q/TNF-related protein levels are associated withreduced arterial stiffness in middle-aged and older adults. Am JPhysiol Regul Integr Comp Physiol 2018; 314: R94–R101.

22. Norroz pour Lari Z, Shemshaki A, Kheirandish M. Effect of eight weeks aerobic interval training (Spinning) on serum level of CTRP3 and some metabolic markers in type 2 diabetes mellitus obese women: Dissertation in M.A: Alzahra university. Faculty of sport sciences. 2017. [in Persian]

23. Mirzendedel Z, attarzadehoseini S, Bijeh N, raouf saeb A A. A Comparison of the Effects of Twelve Weeks Combined Training with Different Ordering on CTRP3, TNF-α, IL6 and Insulin Resistance in Women with Type 2 Diabetes. IJEM. 2019; 21 (4):203-216. [in persian].

24. Yoo HJ, Hwang SY, Hong HC, Choi HY, Yang SJ, Choi DS, et al. Implication of progranulin and C1q/TNF-related protein-3 (CTRP3) on inflammation and atherosclerosis in subjects with or without metabolic syndrome. PloS One 2013; 8(2): e55744.

25. Yoon JG, Kim SH, Rhyu HS. Effects of 16-week spinning and bicycle exercise on body composition, physical fitness and blood variables of middle school students. J Exerc Rehabil 2017; 13(4): 400.

26. Jackson AS, Pollock ML, Ward A. Generalized equations for predicting body density of women. MSSE 1980; 12(3): 175-81.

27. Robergs RA, Landwehr R. The surprising history of the" HRmax= 220-age" equation. J Exerc Physiol Online 2002; 5(2): 1-10.

28. Wilmore J, Costill D. Physiology of Sport and Exercise. 3 [sup] rd ed. Champaign, IL: Human Kinetics. 2005.

29. Zheng Q, Yuan Y, Yi W, Lau WB, Wang Y, Wang X, et al. C1q/TNF-related proteins, a family of novel adipokines, induce vascularrelaxation through the adiponectin receptor-1/AMPK/eNOS/nitric oxidesignaling pathway. Arterioscler Thromb Vasc Biol 2011; 31: 2616–23.

30. Kelly M, Ruderman NB, Tomas E. AMP-activatedprotein kinase and its regulation by adiponectin andinterleukin-6. Scand J Food Nutr 2006; 50: 85-91.

31. Ruan H, Dong LQ. Adiponectin signaling andfunction in insulin target tissues. J Mol Cell Biol 2016; 8: 101-9.

32. Geer EB, Shen W. Gender differences in Geer EB, Shen W. Gender differences ininsulin resistance, body composition, and energy balance. Gend Med 2009;6(1):60–75.

33. Wong GW, Krawczyk SA, Kitidis-Mitrokostas C, Revett T, Gimeno R, Lodish HF. Molecular, biochemical and functional characterizations of C1q/TNF family members: adipose-tissue-selective expression patterns, regulation by PPAR-gamma agonist, cysteine-mediated oligomerizations, combinatorial associations and metabolic functions. Biochem J 2008; 416(2): 161–77

34. Gao C, Zhao S, Lian K. C1q/TNF-related protein 3 (CTRP3) and 9 (CTRP9) concentrations are decreased in patients with heart failure and are associated with increased morbidity and mortality. BMC Cardiovasc Disord, 2019; 19(139): 2-9.

35. Touvra AM, Volaklis KA, Spassis AT, Zois CE, Douda HT, Kotsa K, et al. Combined strength andaerobic training increases transforming growthfactor-1 in patients with type 2 diabetes. Hormones2011; 10: 125-30.

36. Ghafari M, Banitalebi E, Heidari A. Impact of High-Intensity Interval Training and at ConcurrentStrength-Endurance Training on the Levels of SomeAdipokines Associated with Insulin Resistance inWomen with Diabetes Mellitus. Heal Res J 2017; 2: 193-206. [in Persian]

37. Masoodian SM, Toolabi K, Omidifar A, Zabihi H, Rahimipour A, Shanaki M. Increased mRNA Expression of CTRP3 and CTRP9 in Adipose Tissue from Obese Women: Is it Linked to Obesity-Related Parameters and mRNA Expression of Inflammatory Cytokines? Reports of Biochem Mol Biol, 2020; 9(1):71-81.

38. Jorge ML, de Oliveira VN, Resende NM, ParaisoLF, Calixto A, Diniz AL, et al. The effects ofaerobic, resistance, and combined exercise onmetabolic control, inflammatory markers, adipocytokines,and muscle insulin signaling in patientswith type 2 diabetes mellitus. Metabolism 2011; 60: 1244-52.